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Re: hydroxychloroquine and COVID-19 

By: ribit in POPE 5 | Recommend this post (1)
Tue, 07 Apr 20 5:38 AM | 37 view(s)
Boardmark this board | Pope 5 		Msg. 58153 of 62138
(This msg. is a reply to 58152 by Decomposed)
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...years and years ago a kid in my neighborhood got rabies. They put him in a tub at the hospital and packed him in ice. Kept him just about frozen for several days til the fever broke. He lived. No one would suggest that ice cures rabies. What it did was keep the fever from killing him while his body fought it off. Maybe this hydroxychloroquine works the same way against covid 19. It doesn't kill the virus, it just treats the symptoms til your body fights it off. Hydroxychloroquine didn't cure malaria either. It just kept it from killing ya.




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Liberals are like a "Slinky". Totally useless, but somehow ya can't help but smile when you see one tumble down a flight of stairs!


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The above is a reply to the following message:
hydroxychloroquine and COVID-19
By: Decomposed in POPE 5
Tue, 07 Apr 20 5:14 AM 		Msg. 58152 of 62138

I've been doing some reading on how COVID-19 works and have concluded that hydroxychloroquine probably DOES work - or, at least, I can see why it should, particularly when taken with zinc.

The novel coronavirus pneumonia (COVID-19) is an infectious acute respiratory infection caused by the novel coronavirus. The virus is a positive-strand RNA virus with high homology to bat coronavirus. In this study, conserved domain analysis, homology modeling, and molecular docking were used to compare the biological roles of certain proteins of the novel coronavirus. The results showed the ORF8 and surface glycoprotein could bind to the porphyrin, respectively. At the same time, orf1ab, ORF10, and ORF3a proteins could coordinate attack the heme on the 1-beta chain of hemoglobin to dissociate the iron to form the porphyrin. The attack will cause less and less hemoglobin that can carry oxygen and carbon dioxide. The lung cells have extremely intense poisoning and inflammatory due to the inability to exchange carbon dioxide and oxygen frequently, which eventually results in ground-glass-like lung images. The mechanism also interfered with the normal heme anabolic pathway of the human body, is expected to result in human disease. According to the validation analysis of these finds, chloroquine could prevent orf1ab, ORF3a, and ORF10 to attack the heme to form the porphyrin, and inhibit the binding of ORF8 and surface glycoproteins to porphyrins to a certain extent, effectively relieve the symptoms of respiratory distress. Favipiravir could inhibit the envelope protein and ORF7a protein bind to porphyrin, prevent the virus from entering host cells, and catching free porphyrins. Because the novel coronavirus is dependent on porphyrins, it may originate from an ancient virus. Therefore, this research is of high value to contemporary biological experiments, disease prevention, and clinical treatment.

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