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Re: â��Like It Was Designed To Infect Humansâ��: COVID-19 â��Cell Cultureâ�� Theory Gains Steam  

By: ribit in 6TH POPE | Recommend this post (1)
Wed, 27 May 20 8:42 PM | 49 view(s)
Boardmark this board | 6th Edition Pope Board 		Msg. 00792 of 60014
(This msg. is a reply to 00778 by Decomposed)
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...ya obviously knows more about it than I do, so I will appoint you the position of "Pope 6 Fauci".
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Liberals are like a "Slinky". Totally useless, but somehow ya can't help but smile when you see one tumble down a flight of stairs!

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The above is a reply to the following message:
“Like It Was Designed To Infect Humans”: COVID-19 ‘Cell Culture’ Theory Gains Steam
By: Decomposed in 6TH POPE
Wed, 27 May 20 5:47 PM 		Msg. 00778 of 60014

#msg-1133329

Kudos to Nemo for finding this article. The image came from me, though. Professor Petrovsky, in the article, is passing on some bad information and consequently concluding that the virus could be natural.

First, he says the virus could just be a mutation. That would be the case if it had a single altered nucleic acid sequence (the "Letters" you see in DNA and RNA strands represent nucleic acid sequences). If a "G" changed to a "C" in the example I've displayed above ... that could be a natural mutation. But SARS-CoV-2 has four straight ("PRRA" in the image above) that were INSERTED, not changed. Mutations rarely work that way, although it could happen if an enzyme had a wrinkle in it at the time a virus was reproducing. That might result in an insert from some other part of the virus's code. But not in this case, because...

Second, he says “One of the possibilities is that an animal host was infected by two coronaviruses at the same time and COVID-19 is the progeny of that interaction between the two viruses."

Incorrect. The four consecutive sequences which are new to the SARS-CoV-2 virus do not exist in any other coronavirus. They only exist in viruses distantly related (less than a 35% similarity) to SARS-CoV-2.

That also rules out the possibility of a "wrinkle" insert. The odds are overwhelming that this thing was man-made.

And third, the professor says "The way coronavirus enters human cells is by binding to a protein on the surface of lung-cells called ACE2."

That's very old news. They now know it has at least two means of entry, the other being TMPRSS2 if I remember (and understand) correctly. (I'm not strong on pathology.)

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